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that may be, stimulation of CD74 with exogenous MIF may well cause an too much inflammatory condition, as MIF is able to stimulating CXCR2 and CXCR4 receptors Besides CD74. CXCR2 and CXCR4 receptors when activated promote inflow of neutrophils and lymphocytes, respectively (63, sixty four). In contrast, activation of the pro-survival kinase AMPK s